pmcherry c1 plasmid (Addgene inc)
Structured Review
Pmcherry C1 Plasmid, supplied by Addgene inc, used in various techniques. Bioz Stars score: 91/100, based on 5 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/pmcherry+c1/pmCherry-Akt1(WT)+(Plasmid+%2386631)/bio_rxiv__64898__2026__03__31__715582-184-76-78
Average 91 stars, based on 5 article reviews
Images
Related Articles
Construct:Article Title: A single-component optogenetic toolkit for programmable control of microtubule Article Snippet: .. Opto-αTAT1 (αTAT1-mCh-CIBN-NLS-LEXY) was constructed by using the HiFi DNA assembly method. αTAT1, mCherry, CIBN and LEXY (LOV2-NES) were amplified by standard PCR from pEF5B-FRT-GFP-αTAT1 (Addgene, #27099), Article Title: Tau destabilization in a familial deletion mutant K280 accelerates its fibrillization and enhances the seeding effect Article Snippet: .. For mammalian expression constructs, WT tau, mutant tau, and mCherry-eGFP tandem protein were cloned into mammalian expressing vector pEGFP-C1 and Article Title: Tau destabilization in a familial deletion mutant K280 accelerates its fibrillization and enhances the seeding effect. Article Snippet: .. For mammalian expression constructs, WT tau, mutant tau, and mCherry-eGFP tandem protein were cloned into mammalian expressing vector pEGFP-C1 and Amplification:Article Title: A single-component optogenetic toolkit for programmable control of microtubule Article Snippet: .. Opto-αTAT1 (αTAT1-mCh-CIBN-NLS-LEXY) was constructed by using the HiFi DNA assembly method. αTAT1, mCherry, CIBN and LEXY (LOV2-NES) were amplified by standard PCR from pEF5B-FRT-GFP-αTAT1 (Addgene, #27099), Polymerase Chain Reaction:Article Title: A single-component optogenetic toolkit for programmable control of microtubule Article Snippet: .. Opto-αTAT1 (αTAT1-mCh-CIBN-NLS-LEXY) was constructed by using the HiFi DNA assembly method. αTAT1, mCherry, CIBN and LEXY (LOV2-NES) were amplified by standard PCR from pEF5B-FRT-GFP-αTAT1 (Addgene, #27099), Expressing:Article Title: Tau destabilization in a familial deletion mutant K280 accelerates its fibrillization and enhances the seeding effect Article Snippet: .. For mammalian expression constructs, WT tau, mutant tau, and mCherry-eGFP tandem protein were cloned into mammalian expressing vector pEGFP-C1 and Article Title: Tau destabilization in a familial deletion mutant K280 accelerates its fibrillization and enhances the seeding effect. Article Snippet: .. For mammalian expression constructs, WT tau, mutant tau, and mCherry-eGFP tandem protein were cloned into mammalian expressing vector pEGFP-C1 and Mutagenesis:Article Title: Tau destabilization in a familial deletion mutant K280 accelerates its fibrillization and enhances the seeding effect Article Snippet: .. For mammalian expression constructs, WT tau, mutant tau, and mCherry-eGFP tandem protein were cloned into mammalian expressing vector pEGFP-C1 and Article Title: Tau destabilization in a familial deletion mutant K280 accelerates its fibrillization and enhances the seeding effect. Article Snippet: .. For mammalian expression constructs, WT tau, mutant tau, and mCherry-eGFP tandem protein were cloned into mammalian expressing vector pEGFP-C1 and Clone Assay:Article Title: Tau destabilization in a familial deletion mutant K280 accelerates its fibrillization and enhances the seeding effect Article Snippet: .. For mammalian expression constructs, WT tau, mutant tau, and mCherry-eGFP tandem protein were cloned into mammalian expressing vector pEGFP-C1 and Article Title: Tau destabilization in a familial deletion mutant K280 accelerates its fibrillization and enhances the seeding effect. Article Snippet: .. For mammalian expression constructs, WT tau, mutant tau, and mCherry-eGFP tandem protein were cloned into mammalian expressing vector pEGFP-C1 and Article Title: CRTC2 Forms Co-condensates with YTHDF2 that Enhance Translational Efficiency of m6A-Modified mRNAs to Drive Hepatocarcinogenesis and Lenvatinib Resistance Article Snippet: 2 As the third most common cause of cancer-related mortality, hepatocellular carcinoma 36 (HCC) is a global health concern.. Despite its prevalence, treatment options are limited, 37 underscoring the need to identify potential therapeutic targets and strategies.. Here, we 38 identified amplification of CRTC2, situated in the 1q21.3 region, due to copy number 39 alterations in HCC. Fluorescence:Article Title: CRTC2 Forms Co-condensates with YTHDF2 that Enhance Translational Efficiency of m6A-Modified mRNAs to Drive Hepatocarcinogenesis and Lenvatinib Resistance Article Snippet: 2 As the third most common cause of cancer-related mortality, hepatocellular carcinoma 36 (HCC) is a global health concern.. Despite its prevalence, treatment options are limited, 37 underscoring the need to identify potential therapeutic targets and strategies.. Here, we 38 identified amplification of CRTC2, situated in the 1q21.3 region, due to copy number 39 alterations in HCC. other:Article Title: Plasma membrane nanodeformations promote actin polymerization through CIP4/CDC42 recruitment and regulate type II IFN signaling Article Snippet: FBP17 , Human , pmCherry-C1 , CMV , Article Title: Plasma membrane nanodeformations promote actin polymerization through CIP4/CDC42 recruitment and regulate type II IFN signaling Article Snippet: FCHo2 , Mouse , pmCherry-C1 , CMV , Article Title: Plasma membrane nanodeformations promote actin polymerization through CIP4/CDC42 recruitment and regulate type II IFN signaling Article Snippet: FCHo1 , Mouse , pmCherry-C1 , CMV , Plasmid Preparation:Article Title: UVSSA facilitates transcription-coupled repair of DNA interstrand crosslinks. Article Snippet: DNA interstrand crosslinks (ICLs) are covalent bonds between bases on opposing strands of the DNA helix which prevent DNA melting and subsequent DNA replication or RNA transcription.. Here, we show that Ultraviolet Stimulated Scaffold Protein A (UVSSA) is critical for ICL repair in human cells, at least in part via the transcription coupled ICL repair (TC-ICR) pathway.. Inactivation of UVSSA sensitizes human cells to ICL-inducing drugs, and delays ICL repair. |
